Keto Kitchen

06/12/2026

06/11/2026

What If The Problem Was Never Your Willpower?
Bitten Jonsson believes sugar changes the brain long before it changes the body.

For years, I thought people were failing because they lacked discipline.

Then I met people who could quit alcohol, quit smoking, build successful careers, raise families...
..yet could not stop eating sugar.

That made no sense.

Until I discovered the work of Bitten Jonsson.

Her theory is simple:

The problem is not always the food.

The problem is what the food is doing to the brain.

When highly processed sugar and flour hit the reward system, dopamine surges.

For some people, that surge is powerful enough to create a cycle that feels almost impossible to escape.

Cravings arrive.

Energy crashes.

The brain asks for another hit.

Then another.

Then another.

Eventually people begin blaming themselves for a problem that may be far more biological than moral.

Bitten often says that recovery begins when we stop asking:

"Why am I so weak?"

And start asking:

"What is happening inside my brain?"

Her approach focuses first on biochemical healing.

Remove the foods that keep triggering the cycle.

Stabilize blood sugar.

Feed the brain properly.

Allow the reward system time to recover.

Only then can many people begin addressing the deeper emotional layers that often sit underneath years of compulsive eating.

For countless people, that shift changes everything.

Not because they suddenly found more willpower.

Because they finally understood what they were fighting.

06/11/2026

🎗️ What if one of the most powerful tools in cancer prevention starts with improving your metabolic health?

Emerging research continues to highlight the strong connection between insulin resistance, metabolic dysfunction, and cancer risk. It's time to shift the conversation from treating disease to preventing it.

Read our latest blog and discover why metabolic health may be the key to a healthier future:

https://thenoakesfoundation.org/metabolic-health-is-the-new-cancer-prevention/

06/09/2026

One drink. Just one.

Here is exactly what is happening in your liver from the moment that alcohol enters your bloodstream — and why the "one glass of wine is healthy" narrative was one of the most consequential pieces of medical misinformation of the last 30 years.

Within 30 minutes of your first drink, alcohol is absorbed directly through the stomach wall into your bloodstream — faster than almost any other substance you consume. Your liver immediately begins prioritizing its metabolism above every other task it was performing: fat burning stops, hormone regulation is suspended, vitamin activation is paused, toxin filtering from the rest of your body is deprioritized. Everything waits for the alcohol.

The first metabolite your liver produces from alcohol is acetaldehyde — classified by the International Agency for Research on Cancer as a Group 1 carcinogen. This is the compound that causes the flushed feeling, the racing heart, and the nausea. It is directly toxic to DNA — it binds to DNA strands in liver cells and causes the kind of damage that can initiate the mutation cascade leading to cancer. This happens every time you drink. Every single time.

Your liver then converts acetaldehyde to acetate — a less toxic compound — but this conversion consumes NAD+, one of the most critical coenzymes in your entire metabolic system. NAD+ depletion from regular drinking is one of the mechanisms behind accelerated aging, mitochondrial dysfunction, and the cognitive decline associated with chronic alcohol use.

While your liver processes the alcohol — which takes approximately one hour per standard drink — blood sugar crashes because the liver cannot simultaneously regulate glucose and metabolize ethanol. This is why drinking leads to intense food cravings and why the morning after heavy drinking involves profound energy disruption.

The immune suppression window lasts 24 hours after each drinking episode. Every drink is followed by approximately one day of measurably reduced immune function — reduced T-cell activity, reduced natural killer cell output, reduced pathogen surveillance

06/07/2026

🦠 Can the gut microbiome adapt to a carnivore diet?

A new study examining long-term carnivore diet adherents found that while their gut microbiomes differed from those consuming mixed diets, microbial diversity and metabolic functionality were maintained.

The findings challenge the assumption that the absence of plant foods automatically results in poor gut health and suggest that the relationship between diet, fibre, and the microbiome may be more complex than previously thought.

At The Noakes Foundation, we believe science advances when assumptions are tested, evidence is explored, and questions remain open to investigation.

This study adds an interesting perspective to the ongoing conversation about metabolic health, low-carbohydrate nutrition, and the adaptability of the human body.

📖 Read the study:
https://mah.bioscientifica.com/view/journals/mah/4/1/MAH-25-0015.xml

What are your thoughts on these findings?